Why does it make good metabolic sense for
Why does it make good metabolic sense for phosphoenolpyruvate carboxykinase, rather than pyruvate carboxylase, to be the primary target for regulation of gluconeogenesis at the level of control of enzyme synthesis?
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what is the metabolic significance of the following observations 1 only the liver form of pyruvate kinase is inhibited
why does it make good metabolic sense for phosphoenolpyruvate carboxykinase rather than pyruvate carboxylase to be the
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consider an mginfin queue ie a queue with poisson arrivals of rate lambda in which each arrival i independent of other
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