Problem:
Consider the fates of glycogen in both liver and muscle.
Question: What physiological conditions will stimulate glycogen degradation in each tissue?
Question: What are the signals for these conditions, and how do they stimulate glycogen degradation?
Question: What would the major consequence be of the loss of the AMP binding site in glycogen phosphorylase?
Question: What would the major consequence be of a mutation of Ser14 to Ala14 in glycogen phosphorylase?
Could someone help to explain the answer of these questions.