Many eukaryotic genes have two stop codons located close to each other at the end of the gene. How might this be relevant for cells that carry a nonsense suppressor mutation?
List at least three ways that concentrations of a particular functional mRNA can be regulated.
It can be regulated by binding to proteins, which recognizes the sequence in the mRNA. In most cases the protein act to inhibit translation.
Considering spontaneous mutations caused by depurination, deamination, and tautomeric shifts, explain why transitions are more common than transversions.
Why are high copy cloned genes likely to be silenced?
What might be a functional advantage to more efficient repair of actively transcribed genes?