Problem
A. According to the Oxford Handbook of Clinical Medicine, an NEJM trial for the treatment of osteoarthritis failed with glucosamine and chondroitin sulfate?Are these compounds still useful in any way?
B. In established rheumatoid arthritis that is in remission, can high serum titres of C-reactive protein (CRP) with normal erythrocyte sedimentation rate (ESR) indicate relapse?
C. In the diagnosis of gout, what is the positive predictive value of hyperuricaemia? What exactly is Scheuermann's disease?And is this a particular hematological, clinical, or radiological condition?How is the disease treated?
D. How is the body weight-based plasma creatinine clearance value determined at the bedside?
E. Could you tell me if it is still considered effective to administer low doses of dopamine to increase renal blood flow?
F. Why does anuria result from hemoglobinuria?
G. The use of a "high-protein diet" to treat nephrotic syndrome baffles me.You claim that it has no benefit, but the Oxford Handbook of Clinical Medicine recommends using it, and Davidson's Principles and Practice of Medicine states that it is even risky because it may damage the kidneys.If I see the question in an MCQ, which one should I select?
H. Nephrotic syndrome: Is albumin infusion contraindicated?What are the signs if this is not the case?
I. It has been stated that high doses of captopril can cause immune-complexmediated membranous glomerulonephritis, which is one of the drug causes of nephrotic syndrome.Is it harmful to administer captopril as a hypertension treatment to a patient with nephrotic syndrome?Could this aggravate the nephrotic syndrome of the patient?