Discussion:
Neuromuscular blocking agents and transmembrane potential gradients
1. A certain type of poison is a nicotinic receptor blocker. What affect would this poison have on the muscle action potential?
2. Some inhibitory postsynaptic potentials (IPSPs) are the result of Cl- entering the cell through a chloride channel. Other IPSPs are caused by the opening of K+ channels. How could movement of the potassium ion result in hyperpolarization of the postsynaptic cell.
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