Type-1 mutations in the head domain of kif5a have defects


Type-1 mutations in the head domain of KIF5A have defects in the binding of the protein to tubulin. How would weakened binding to tubulin be expected to affect the function of kinesin and the transport of vesicles in neurons? How would overly strong binding to tubulin be expected to affect the function of kinesin? Please limit your answer to the lines below

Solution Preview :

Prepared by a verified Expert
Biology: Type-1 mutations in the head domain of kif5a have defects
Reference No:- TGS02702236

Now Priced at $10 (50% Discount)

Recommended (94%)

Rated (4.6/5)