A second woman has breast cancer, but her tumor cells have normal levels of estrogen receptors. Instead, they have G proteins that are constantly active, producing high levels of the second messenger cyclic AMP.
How could this cause the cells to grow?
Which of the three treatments in question #2 probably would not work well for this woman?
Why? Treatments in #2: Tamoxifen, An antibody to the tumor cells with a toxin attached to it, Lumpectomy followed by local radiation
If you have any links that would lead me to answer this question, that would be great!!